Introduction Patients with type 2 diabetes have increased blood sugar levels. High blood glucose in type 2 diabetes may be caused by inadequate insulin being produced by the pancreas to control blood sugar, by a decrease in the effects of insulin on peripheral tissues, or by the effect of both factors together . A dysregulation of the oxidative and glycolytic capacities of skeletal muscle also leads to insulin resistance in type 2 diabetes. The disease most likely results from increased body weight, lack of physical activity, consumption of unhealthy foods, blood pressure high or by increased cholesterol levels and age. People over the age of 40 are at greater risk of type 2 diabetes, but the disease is now becoming more common in children, adolescents and young adults. Furthermore, the likelihood of developing type 2 diabetes is also influenced by genetics. The disease can be recognized by its symptoms such as increased thirst and frequent urination because the high blood sugar level causes fluid to leak from the tissues. This may make the patient thirsty. As a result, the patient may drink and urinate more frequently. Increased hunger is one of the symptoms of type 2 diabetes when without enough insulin to move sugar into cells, muscles and organs run out of energy. This makes the patient feel hungry. Fatigue is also a very common symptom because the cells are deprived of sugar, the patient becomes tired and irritable. Additionally, blurred vision is also common among individuals with type 2 diabetes. High blood sugar can cause fluid to leak from the lenses of the eyes. This may affect your ability to concentrate clearly. Slow-healing sores, frequent infections, and darkened skin may also appear in individuals with type 2 diabetes. This disease...... middle of paper...... physical activity, increases oxygen consumption throughout the body . Skeletal muscle uses, at a significantly increased rate, its stores of glycogen, triglycerides and free fatty acids (FFA) derived from the breakdown of triglycerides from adipose tissue and glucose released by the liver. Central nervous system function is preserved by remarkably maintained blood glucose levels during physical activity. Hypoglycemia during exercise does not often occur in non-diabetic individuals. The metabolic adjustments that maintain normoglycemia during exercise are largely hormone-mediated. A reduced plasma insulin level and the presence of glucagon appear to be necessary during physical activity. In patients with type 2 diabetes, physical activity can improve insulin sensitivity and help reduce high blood glucose levels to within normal ranges.